Track Categories

The track category is the heading under which your abstract will be reviewed and later published in the conference printed matters if accepted. During the submission process, you will be asked to select one track category for your abstract.

 The pancreatic cancer starts with the tissue of pancreas an organ in the abdomen that lies horizontally behind the lower part of the stomach. Pancreas mainly releases the hormones and enzymes that aid the digestion as well as blood sugar level. The pancreatic cancer easily spreads to other parts of the body. These changes are rarely unseen and sometimes undetectable before it becomes severe in kind. This might be detectable in the situations like, family history of cancer or pancreatic cysts. However to people with pancreatic cysts or a family history from claiming pancreatic cancer some screening steps might assistance recognize an issue right on time. Person sign of pancreatic tumor may be diabetes, particularly when it happens for weight loss, jaundice alternately ache in the upper abdomen that spreads of the back. Medication might incorporate surgery, chemotherapy, radiation help alternately a consolidation of these.

The main aim of the diabetes is to detect the overall condition of the disorder, there are many case research studies including animal models and human models. The integrated care for diabetes mellitus in terms of the intermediate clinical and process outcomes, the evidence-based knowledge on its implementation is scarce, and insights generalizable to other settings therefore remain limited.  This study protocol provides a description of the design and methodology of a mixed methods study on the implementation of integrated care for diabetes. The aim of the proposed research is to investigate the mechanisms by which and the context in which integrated care for diabetes has been implemented, which outcomes have been achieved and how the context and mechanisms have affected the outcomes.

Diabetes can cause early onset and increased severity of these problems. Sexual and urologic complications of diabetes occur because of the damage diabetes can cause to blood vessels and nerves. Men may have difficulty with erections or ejaculation. Women may have problems with sexual response and vaginal lubrication. Urinary tract infections and bladder problems occur more often in people with diabetes. People who keep their diabetes under control can lower their risk of the early onset of these sexual and urologic problems. Both men and women with diabetes can develop sexual problems because of damage to nerves and small blood vessels. A number of clinical studies in men and women with diabetes have reported bladder instability or hypersensitivity as the most frequent finding, ranging from 39–61% of subjects.

Nephropathy means kidney disease or damage. Diabetic nephropathy is damage to your kidneys caused by diabetes. In severe cases it can lead to kidney failure. But not everyone with diabetes has kidney damage.The kidneys have many tiny blood vessels that filter waste from your blood. High blood sugar from diabetes can destroy these blood vessels. Over time, the kidney isn't able to do its job as well. Later it may stop working completely. This is called kidney failure. Certain things make you more likely to get diabetic nephropathy. If you also have high blood pressure or high cholesterol, or if you smoke, your risk is higher. Also, Native Americans, African Americans, and Hispanics (especially Mexican Americans) have a higher risk.

The Complications in diabetes is discussing about that the struggle and severe effects, and harmfulness of affecting parts of body due to diabetes.  Long-standing complications of diabetes grow progressively which shows that the number of years of having diabetes is directly proportional to the higher risk of complications. Sometimes diabetes complications may be restricting or even lethal. Focusing on diabetic complications is important. An Controlled blood sugar have less diabetic complications. If the blood sugar level has not maintained properly means it will have more diabetic complications. The briefer ideas of health issues quicken the venomous effects of diabetes which include smoking, high diabetes, high cholesterol levels, obesity, high blood pressure, and lack of exercise. The process by which vascular disease develops is complex and occurs via numerous pathways that scientists continue to investigate.

  • Track 5-1Diabetic Nephropathy
  • Track 5-2Diabetic Neuropathy
  • Track 5-3Diabetic Retinopathy

Diabetes Insipidus occurs when the kidneys cannot concentrate the urine normally, and a large amount of dilute urine is excreted. The amount of water excreted in the urine is controlled by antidiuretic hormone (ADH). ADH is also called vasopressin. ADH is produced in a part of the brain called the hypothalamus. It is then stored and released from the pituitary gland. DI caused by a lack of ADH is called central diabetes insipidus. When DI is caused by a failure of the kidneys to respond to ADH, the condition is called nephrogenic diabetes insipidus.

Diabetes epidemiology is to describe and analyse the causes and consequences of the pandemic of type 1 and type 2 diabetes, and to develop and test strategies that might help to put this pandemic into reverse.

Understanding the pathogenesis of type 2 diabetes is complicated by several factors. Patients present with a combination of varying degrees of insulin resistance and relative insulin deficiency, and it is likely that both contribute to type 2 diabetes. Furthermore, each of the clinical features can arise through genetic or environmental influences, making it difficult to determine the exact cause in an individual patient. Moreover, hyperglycemia itself can impair pancreatic beta-cell function and exacerbate insulin resistance, leading to a vicious cycle of hyperglycemia causing a worsening metabolic state.

Diabetes mellitus is a heterogeneous group of disorders characterized by persistent hyperglycemia. The two most common forms of diabetes are type1 diabetes (T1D, previously known as insulin-dependent diabetes or IDDM) and type 2 diabetes (T2D, previously known as non-insulin-dependent diabetes or NIDDM). Both are caused by a combination of genetic and environmental risk factors. However, there are other rare forms of diabetes that are directly inherited. These include maturity onset diabetes in the young (MODY), and diabetes due to mutations in mitochondrial DNA.

Diabetes is a disease in which your blood glucose, or blood sugar, levels are too high. Insulin is a hormone that helps the glucose get into your cells to give them energy. With type 1 diabetes, your body does not make insulin. With type 2 diabetes, the more common type, your body does not make or use insulin well. Without enough insulin, the glucose stays in your blood. You can also have prediabetes. This means that your blood sugar is higher than normal but not high enough to be called diabetes. Having prediabetes puts you at a higher risk of getting type 2 diabetes.

  • Track 10-1Metabolism disorder.
  • Track 10-2Causes
  • Track 10-3Symptoms

Diabetes mellitus is the most common endocrine metabolic disorder that is multifactorial such as, genetic factor and environment elements. Basically our body breaks down the sugar and carbohydrates into simple sugar as glucose. This glucose is required to the body cells in order to activate the metabolism. This job is done by the hormone called Insulin, which produced by beta cells. The person with high blood sugar either will have insulin resistance in the body or deficit production of insulin. The patients with high blood sugar will have symptoms such as, frequent of urination, more thirsty and hungry. Because of such factors, the glucose will start to deposit on blood vessels in heart, kidney which will damage the parts. It can eventually causes heart diseases, kidney diseases and nerve system damage.

  • Track 11-1Type 1 diabetes
  • Track 11-2Type 2 diabetes
  • Track 11-3Gestational diabetes

Diabetes may be analysed based on both plasma glucose criteria or  A1C criteria. Early in disease diabetes shows no symptoms so blood tests are used to diagnose diabetes and pre-diabetes. Clinical diagnostic in diabetes involve taking blood at a healthcare facility centre or commercial diagnostic centre and sending the sample to a lab for examination. To ensure that test results are accurate lab study of blood of required. Glucose calculating devices used in a health care provider’s office, such as finger-stick devices, are not precise enough for analysis but may be used as a rapid indicator of high blood sugar. Such area of research are highle discussed in  ADA annual meeting  and ADA sessions.

One of the most important factors in the development of diabetes is genetics (over which we have no control). However, there are things in our environment and lifestyle we can control to lower our personal risk of developing diabetes, including exercise and weight loss, avoiding smoking, diet control.

  • Track 13-1Physical Activity
  • Track 13-2Oral medications or insulin
  • Track 13-3Target HbA1c level
  • Track 13-4Diet control

Diabetes is a genuine ailment that you can't treat all alone. Your specialist will enable you to make a diabetes treatment arrange for that is appropriate for you and that you can get it. You may likewise require other human services experts on your diabetes treatment group, including a foot specialist, nutritionist, eye specialist, and diabetes pro called an Endocrinologist. Treatment for diabetes requires keeping close watch over your glucose levels and keeping them at an objective set by your specialist with a mix of pharmaceuticals, exercise, and eating regimen. Diabetes is a typical illness, yet every individual needs remarkable care. We energize individuals with diabetes and their families to learn however much as could reasonably be expected about the most recent therapeutic treatments and methodologies, and additionally solid way of life decisions. Great correspondence with a group of specialists can enable you to feel in charge and react to evolving needs.

Diabetes mellitus DM), a condition of constant hyperglycaemia, is a noteworthy reason for genuine small scale and macro-vascular infections, influencing, hence, almost every framework in the body. Developing proof shows that oxidative anxiety is expanded in diabetes because of overproduction of receptive oxygen species (ROS) and diminished proficiency of cancer prevention agent barriers, a procedure that begins early and intensifies through the span of the infection. Amid the advancement of diabetes, oxidation of lipids, proteins and DNA increment with time. Mitochondrial DNA changes have likewise been accounted for in diabetic tissues, proposing oxidative anxiety related mitochondrial harm. Diabetes-related oxidative anxiety may likewise be the trigger for some adjustments on sexual capacity, which can likewise incorporate diminished testicular mitochondrial work. Albeit sexual clutters have been broadly examined in diabetic men, conceivable changes in the sexual capacity of diabetic ladies have just as of late gotten consideration. The commonness of sexual brokenness in diabetic men approaches half, though in diabetic ladies it is by all accounts marginally lower.

Diabetes is a disorder which can be caused due to many factors among them immunity is also one of the factors. The destruction of the islet β-cell in T1D is the result of a  complex interplay between multiple players of both the innate and adaptive immune system; immunohistochemical analysis of islet inflammation from pancreata of patients with T1D obtained at autopsy indicate a mononuclear cell infiltrate in islets (termed insulitis) consisting  mainly of macrophages, B cells and T cells. Both CD4+ and CD8+ T cells are required for disease development, by destroying the insulin-producing β cells through the effector functions of Th1 cells and direct killing by cytotoxic T lymphocytes (CTLs). CTLs initiate killing by various mechanisms including the production of inflammatory cytokines such as TNF-a and IFN-g, which act synergistically with IL-1 produced by macrophages in targeting the β-cells; they also directly kill β-cells through the secretion of perforin or by apoptosis by the activation of the Fas-Fas-L pathway.